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Rebecca Voelker
JAMA contributor

JAMA. 1998;279:1245-1246.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

A HALF CENTURY AGO, it would have been unthinkable.

Take a traditional epidemiologic study aimed at finding the causes of cardiovascular disease and focus the search on the very genes that cause some people to be afflicted with hypertension and other conditions while others go through life never having to give their blood pressure or diet a second thought?

As the celebrated Framingham Heart Study passes the 50-year landmark, researchers analyzing its rich data collections have taken a definite turn to molecular genetics.

"We have come to the realization that in looking at risk factors for disease—smoking, high blood pressure—that are largely lifestyle dependent, they do not explain the whole story," said Claude Lenfant, MD, director of the National Heart, Lung, and Blood Institute, which sponsors the Framingham Study.

"We began the process of collecting blood samples for DNA extraction about 8 or 9 years ago," said Daniel . . . [Full Text of this Article]

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