 |
|
Methylmercury Exposure and Neurotoxicity
Kathryn R. Mahaffey, PhD
JAMA. 1998;280:737-738.
|
|
| Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings. |
|
|
|
Exposure to high levels of methylmercury has produced fatalities and devastating neurological damage among adult survivors.1 Severe cases of in utero methylmercury poisoning strongly resemble cerebral palsy and are often accompanied by deafness and mental retardation.1-2 In addition, clinically evident cases of neurological damage have occurred among infants born to mothers whose own symptoms were often only transient paresthesias. Consequently, the fetus is considered much more sensitive to methylmercury exposure than is the adult.
Although recognizing clinically evident symptoms is relatively straightforward, determining levels of in utero methylmercury exposures associated with the onset of toxic responses in infants and children is a much more complex problem. In this issue of THE JOURNAL, Davidson et al3 present findings from a cohort in the Republic of Seychelles that indicate consumption of methylmercury from fish did not produce developmental deficits among children exposed both in utero and postnatally to . . . [Full Text of this Article]
From the National Center for Environmental Assessment, US Environmental Protection Agency, Washington, DC.
 CiteULike  Connotea  Del.icio.us  Digg  Reddit  Technorati  Twitter
What's this?
RELATED ARTICLE
Effects of Prenatal and Postnatal Methylmercury Exposure From Fish Consumption on Neurodevelopment: Outcomes at 66 Months of Age in the Seychelles Child Development Study
, , , , , , , , , , , and
JAMA. ;280():701-707.
FULL TEXT
|
