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Alan H. Jobe, MD,PhD

JAMA. 2001;285:2244-2245.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Sudden infant death syndrome (SIDS) is one of the most tragic events in medicine. The major hypotheses for the causes of SIDS are variants of the presumption that the SIDS event results from abnormal cardiopulmonary regulation, perhaps aggravated by environmental factors. Although multiple "immaturities" or "abnormalities" of cardiorespiratory control in preterm, term, and young infants have been demonstrated,¹ the links between those physiological responses and SIDS are tenuous. Nevertheless, a home monitoring industry with the noble goal of preventing SIDS has now operated for many years without either a sound scientific foundation for monitoring or a clear demonstration that monitoring decreases SIDS.

Based on epidemiologic information that demonstrated lower rates of SIDS in Asian vs American populations, the practice of placing infants prone for sleep was identified as a risk factor for SIDS.² The US custom of a prone sleeping position for infants was thought to . . . [Full Text of this Article]

Author Affiliation: Division of Pulmonary Biology, Children's Hospital Medical Center, Cincinnati, Ohio.

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