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Loop Diuretics for Patients With Acute Renal Failure
Helpful or Harmful?
Norbert Lameire, MD, PhD;
Raymond Vanholder, MD, PhD;
Wim Van Biesen, MD, PhD
JAMA. 2002;288:2599-2601.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings. |
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Intrinsic acute renal failure (ARF) that does not result from primary vascular, glomerular, or interstitial disorders has been ascribed to acute tubular necrosis and accounts for approximately 45% of cases of hospital-acquired ARF.1 While acute tubular necrosis usually is caused by ischemic (50%) or nephrotoxic (35%) injury to the kidney,2 the cause is often multifactorial. Unfortunately, neither the occurrence of acute tubular necrosis nor the morbidity and mortality associated with it have declined despite ongoing improvement in the supportive care of patients with renal failure and the advent and availability of intermittent and continuous dialysis.3-4 One reason for this lack of improvement is a change in the severity of the underlying diseases causing ARF.1, 4-5
The incidence of acute tubular necrosis is particularly high in patients admitted to an intensive care unit (ICU). The spectrum of acute tubular . . . [Full Text of this Article]
Author Affiliations: Renal Division, University Hospital, Ghent, Belgium.
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