You are seeing this message because your Web browser does not support basic Web standards. Find out more about why this message is appearing and what you can do to make your experience on this site better.


ABOUT JAMA
Advanced Search

Welcome   | My Account | E-mail Alerts | Access Rights | Sign In


  Vol. 292 No. 19, November 17, 2004 TABLE OF CONTENTS
  JAMA
  •  Online Features
  JAMA Patient Page
 This Article
 •Full text
 •PDF
 •Spanish PDF
 •Send to a friend
 • Save in My Folder
 •Save to citation manager
 •Permissions
 Citing Articles
 •Contact me when this article is cited
 Related Content
 •Related articles
 •Similar articles in JAMA
 Topic Collections
 •Cardiovascular System
 •JAMA Patient Page
 •Cardiovascular Disease/ Myocardial Infarction
 •Alert me on articles by topic
 Social Bookmarking
  Add to CiteULike Add to Connotea Add to Del.icio.us Add to Digg Add to Reddit Add to Technorati Add to Twitter What's this?

Left Ventricular Hypertrophy

Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

The major pumping chamber of the heart is the left ventricle. This heart chamber pumps oxygenated blood into the aorta, the large blood vessel that delivers blood to the body's tissues. If the left ventricle has to work too hard, its muscle hypertrophies (enlarges) and becomes thick. This is called left ventricular hypertrophy (LVH). Because of the increased thickness, blood supply to the muscle itself may become inadequate. This can lead to cardiac ischemia (not enough blood and oxygen at the tissue level), myocardial infarction (heart attack), or heart failure. The November 17, 2004, issue of JAMA includes several articles about reducing the risks of heart failure and death from LVH by treating high blood pressure.


CAUSES OF LVH

  • Hypertension (high blood pressure)
  • Obesity
  • Aortic valve stenosis (narrowing of the valve from the heart to the aorta)
  • Obstructive cardiomyopathy (an inherited type of LVH that slows blood flow to the aorta . . . [Full Text of this Article]

Janet M. Torpy, MD, Writer; Tiffany J. Glass, MA, Illustrator; Richard M. Glass, MD, Editor



Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us   Add to Digg Digg   Add to Reddit Reddit   Add to Technorati Technorati   Add to Twitter Twitter     What's this?

RELATED ARTICLES

Regression of Electrocardiographic Left Ventricular Hypertrophy During Antihypertensive Treatment and the Prediction of Major Cardiovascular Events
Peter M. Okin, Richard B. Devereux, Sverker Jern, Sverre E. Kjeldsen, Stevo Julius, Markku S. Nieminen, Steven Snapinn, Katherine E. Harris, Peter Aurup, Jonathan M. Edelman, Hans Wedel, Lars H. Lindholm, Björn Dahlöf, and for the LIFE Study Investigators
JAMA. 2004;292(19):2343-2349.
ABSTRACT | FULL TEXT  

Prognostic Significance of Left Ventricular Mass Change During Treatment of Hypertension
Richard B. Devereux, Kristian Wachtell, Eva Gerdts, Kurt Boman, Markku S. Nieminen, Vasilios Papademetriou, Jens Rokkedal, Katherine Harris, Peter Aurup, and Björn Dahlöf
JAMA. 2004;292(19):2350-2356.
ABSTRACT | FULL TEXT  






HOME | CURRENT ISSUE | PAST ISSUES | TOPIC COLLECTIONS | CME | SUBMIT | SUBSCRIBE | HELP
CONDITIONS OF USE | PRIVACY POLICY | CONTACT US | SITE MAP
 
© 2004 American Medical Association. All Rights Reserved.