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JAMA. 2008;300(17):2044-2045.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

In a Clinical Crossroads article published in April 2006,¹ Gordon J. Strewler, MD, discussed the epidemiology and pathophysiology of primary hyperparathyroidism, as well as the medical and surgical treatment options. The patient, Ms Q, had a history of hypercalcemia first noted on random testing in 2001. Her initial calcium level was 11.3 mg/dL, and her parathyroid hormone level was elevated at 94 pg/mL. A year later, at the time of the conference, her calcium level was 10.4 mg/dL, with albumin at 4.1 g/dL, phosphate at 3.4 mg/dL, and parathyroid hormone at 101 pg/mL. In 2000, her bone density was normal (T score, –0.88 spine and –0.09 total hip). Two years later, her bone density had decreased by 7.3% at the spine and 5.2% at the femoral neck. Based on these findings, Ms Q was believed to have mild primary hyperparathyroidism and was referred to a parathyroid surgeon for consideration of . . . [Full Text of this Article]

MS Q

Nadine Farag, BS; Tom Delbanco, MD; Gordon J. Strewler, MD

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