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Hyperlipoproteinemia
H. R. Casdorph, MD, PhD
Long Beach, Calif
JAMA. 1969;207(1):151.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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To the Editor:—
After reading the letter entitled "Hyperlipoproteinemia—A Difference of Opinion" (206:375, 1968), I should like to add support to the article of Fallon and Woods (204:1161, 1968). Perhaps the criticisms by Stanley Schor, PhD, were stimulating, but I cannot agree with them.
In their article, Fallon and Woods applied statistical evaluation to the response to each individual in part because they represented different types of inherited defects as shown by the type of hyperlipoproteinemia classified by the method of Fredrickson and Lees. The available evidence indicates that these are different inherited defects and it is certainly valid to deal with them as different entities even though they have as a common denominator elevation of their blood lipids. The authors clearly state that 14 patients with essential hypercholesterolemia (type II hyperlipoproteinemia) responded with a 24% decline in serum cholesterol level. Seven patients with other forms of hyperlipoproteinemia had
. . . [Full Text PDF of this Article]
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