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A Dominant Symptom in Diabetes Insipidus

Sandra B. Raff, MD; Herbert Gershberg, MD

JAMA. 1975;234(12):1252-1253.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

A PATIENT had night sweats following an intracranial infection. The sweats were not accompanied by nocturia and were believed to be caused by recurrence of infection. The diagnosis of diabetes insipidus was subsequently established and the night sweats responded to injections of pitressin. The absence of nocturia was attributable to dilatation of the urinary collecting system.

Report of a Case

A 10-year-old boy was admitted to Long Island Jewish Hospital complaining of lethargy, visual hallucinations, and neck pain. He was transferred to New York University Hospital because of decreasing sensorium. On arrival, he had low-grade fever, lack of response to painful stimuli, generalized lymphadenopathy, and signs of cerebral dysfunction (right greater than left) with no papilledema. The cerebrospinal fluid was lymphocytic, with no organisms seen on microscopic examination. There was moderate peripheral leukocytosis, but cultures of all blood, urine, and cerebrospinal fluid specimens were normal. Carotid angiography was compatible with . . . [Full Text PDF of this Article]

Author Affiliations
From the Department of Medicine, New York University School of Medicine, New York.

Footnotes
Reprint requests to Department of Medicine, New York University School of Medicine, 550 First Ave, New York, NY 10016 (Dr Gershberg).

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