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Spontaneous Rupture of the Spleen in Homozygous Hemoglobin C Disease
Mark Lipshutz, MD;
David A. McQueen, MD;
Fred Rosner, MD
JAMA. 1977;237(8):792-793.
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SPONTANEOUS or pathologic rupture of the spleen has been reported in a variety of clinical conditions.1 We report a case of spontaneous splenic rupture in a patient with homozygous hemoglobin C disease.
Report of a Case
A 25-year-old man was hospitalized with an 18-hour history of unremitting abdominal pain located below the umbilicus and associated with two episodes of syncope. There was no history of abdominal trauma, anemia, or jaundice.
The patient was well developed and well nourished. Blood pressure was 120/80 mm Hg, without orthostatic change; pulse was 104 beats per minute and the temperature was 37.2 C. Pertinent physical findings included nonicteric sclerae, normal fundi, lungs that were clear to percussion and auscultation, and a hyperdynamic cardiac impulse with a grade 2/6 systolic precordial murmur. There was no hepatosplenomegaly, nor were any masses felt. Voluntary and involuntary muscular guarding with generalized rebound tenderness was present. Bowel sounds
. . . [Full Text PDF of this Article]
Author Affiliations
From the Division of Hematology, Queens Hospital Center, Affiliation of the Long Island Jewish-Hillside Medical Center, Queens, NY, and from the Health Sciences Center, State University of New York, Stony Brook, NY. Dr McQueen is now with Mt Sinai Hospital Services, City Hospital Center at Elmhurst, Flushing, NY.
Footnotes
Reprint requests to Division of Hematology, Queens Hospital Center, Affiliation of the Long Island Jewish-Hillside Medical Center, 82-68 164th Street, Jamaica, NY 11432.
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