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Abraham Weinberger, MD
University of Pennsylvania School of Medicine Philadelphia

JAMA. 1979;241(8):790.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

Eason et al (240:670, 1978) recently described dissolution of bilateral uric acid lithiasis in a patient with anuria. This patient had a serum uric acid level of 11.5 mg/dL and a creatine level of 6.5 mg/dL. The authors assumed that their patient had uric acid calculi; however, a radiolucent defect was observed on the patient's pyelogram, which might have been due to cystine lithiasis as well as uric acid stones. The conservative treatment for both conditions is the same as was shown in The Journal by my colleagues and me (229:1045, 1974).

The transient high serum uric acid level of the described patient could have been a result of the acute renal failure, and that it returned to normal values was not due to the allopurinol effect but because diuresis began. After dissolution of the renal stones and when the patient is no longer receiving allopurinol, one . . . [Full Text PDF of this Article]

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