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Valentine J. Burroughs, MD; Terry F. Seltzer, MD; Manfred Blum, MD
New York University Medical Center New York

JAMA. 1981;246(1):39-40.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

It is important for therapeutic as well as academic reasons to identify patients who have the newly described silent, or painless, thyroiditis and to differentiate them from patients with hyperthyroidism of other cause. The former condition is usually self-limiting, occasionally recurring, and rarely may lead to long-term hyperthyroidism or hypothyroidism. Symptomatic treatment is all that is usually required, and methods that destroy thyroid tissue or interfere with hormone production should not be employed. Elevated concentration of thyroid hormone, a low radioactive iodine uptake (RAIU), and a normal ESR are prerequisites for a reliable diagnosis of the condition. However, ingestion of thyroid hormone or an expanded iodine pool also reduces RAIU and therefore can confuse the diagnosis. Subacute thyroiditis can usually be differentiated by the high ESR and on clinical grounds.

Recently, Check and Avellino (1980; 244:1361) reported as an example of painless thyroiditis and transient thyrotoxicosis after . . . [Full Text PDF of this Article]

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