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Susumu Kanzaki, MD; Tsutomu Omura, MD; Masako Miyake, MD; Sumiko Enomoto, MD; Ichiro Miyata, MD; Hiroshi Ishimitsu, MD

JAMA. 1985;254(23):3349-3350.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

NEPHROGENIC diabetes insipidus (NDI) is a congenital hereditary disorder characterized by insensitivity of the renal tubule to antidiuretic hormone. This congenital hereditary disorder has been associated with a number of central nervous system complications.^1,2 However, intracranial calcification is an extremely rare complication of this disorder.³ We describe a 2-year-old boy with NDI who presented with a unique pattern of intracranial calcification. Our case shows that NDI may result in organic brain damage if treatment is delayed.

Report of a Case

A boy 2 years 11 months old was admitted to our hospital because of a seizure. He was the product of a normal pregnancy, labor, and delivery of a 30-year-old, gravida 3, para 1, abortus 2 woman. The parents are not related and are of Japanese extraction. His birth weight was 2,900 g. He was given an Apgar score of 9 at both one and five minutes after . . . [Full Text PDF of this Article]

Author Affiliations
From the Departments of Pediatrics (Drs Kanzaki, Omura, Miyake, and Enomoto) and Neurosurgery (Drs Miyata and Ishimitsu), Iwakuni (Japan) National Hospital.

Footnotes
Reprint requests to Department of Pediatrics, Iwakuni National Hospital, Kuroiso-cho, 2-5-1, Iwakuni 740, Japan (Dr Kanzaki).

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