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Edward R. Blonz, PhD

JAMA. 1986;256(22):3138-3139.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THERE IS a growing underground of public controversy surrounding the reputed presence of a "new epidemic." This epidemic involves chronic candidiasis, a condition in which there is an overgrowth of and systemic invasion by the yeast organism Candida albicans.

The Candida organism is ubiquitous in our environment, is a normal inhabitant of the large intestine, and is typically the identifiable organism in vaginal yeast infections (vulvovaginal candidiasis). Of late, a growing popular connotation of "candidiasis" is no longer limited to the female malady. It has come to signify a chronic condition with a new panoply of symptoms.

Perhaps the original proponent of this popular Candida theory is C. O. Truss, MD, of Birmingham, Ala.¹ His hypothesis states that a number of conditions, such as an overuse of antibiotics, will decrease Candida's naturally occurring competitors in the large intestine. This creates an imbalance and facilitates an overgrowth of the Candida . . . [Full Text PDF of this Article]

Author Affiliations
From the Department of Food Science and Nutrition, University of Minnesota, St Paul.

Footnotes
Reprint requests to Department of Food Science and Nutrition, University of Minnesota, 1334 Eckles Ave, St Paul, MN 55108 (Dr Blonz).

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