This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Contact me when this article is cited  Related Content  • Similar articles in JAMA  Social Bookmarking   What's this?

Isaac Samuel, MB
Tiruvella Medical Mission India

JAMA. 1987;258(10):1329-1330.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

Kartagener's syndrome (chronic sinopulmonary symptoms and situs inversus) was shown to be a part of the immotile-cilia syndrome (chronic sinopulmonary symptoms and male sterility) when Afzelius and coworkers demonstrated that males with the immotile-cilia syndrome had immotile (or dysmotile) respiratory cilia and spermatozoa and that 50% of cases of the immotile-cilia syndrome had situs inversus.^1,2 The immotility is attributed to an ultrastructural defect of the respiratory cilium and sperm tail.¹ The inheritance is autosomal recessive. Absence of frontal sinuses is a recognized association.²

This is to my knowledge the second report of a case of Kartagener's syndrome with normal spermatozoa. The patient had chronic sinopulmonary symptoms, situs inversus, and absent frontal sinuses, while his spermatozoa had normal motility, ultrastructure, and fertilizing capacity. Thus, the entity can be regarded as a new subgroup of the immotile-cilia syndrome.

Report of a Case.—

A bachelor, aged 26 . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?