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Brian R. Kaye, MD; Anne Andrews Louis, MD; Molly Fainstat, MD
Santa Clara Valley Medical Center San Jose, Calif Stanford (Calif) University School of Medicine

JAMA. 1988;259(11):1649.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In Reply.

—We would like to thank Drs Levine, Welch, and Brust for raising the interesting possibility of subarachnoid hemorrhage as the etiology for the neurological events that occurred in our patient. Certainly, subarachnoid hemorrhage, arteriovenous malformations, cerebral ischemia, and cerebral vasculitis can all present with similar clinical manifestations.

It is unfortunate that the cerebrospinal fluid obtained from our patient was not examined for xanthochromia because it makes it difficult to know whether the blood in the cerebrospinal fluid was the result of a traumatic spinal tap. We also should note that the left-sided cerebral circulation, as mentioned in our article, was normal. The distal vessels of the right-sided cerebral circulation did not fill with dye.

Although the cerebral angiographic findings in our patient could be the result of vasospasm following a subarachnoid hemorrhage, there are several radiographic features that make this diagnosis less likely. First of all, in patients . . . [Full Text PDF of this Article]

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