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Dennis D. Venable, MD
Louisiana State University Medical Center Shreveport

JAMA. 1988;259(22):3269.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—In a recent letter, Chaudhuri et al¹ reported their anecdotal experience with a single case of presumed diabetic cystoparesis in which their patient demonstrated "improved bladder function" after ten days of oral administration of metoclopramide hydrochloride at a daily dosage of 40 mg. The metoclopramide treatment was administered for diabetic gastroparesis, and the improved bladder function was evidenced by a decrease in an asymptomatically elevated postvoid residual urine volume.

Nestler et al² had earlier published a similar case report of improvement in bladder function in a diabetic man who had required intermittent catheterization and subsequently resumed spontaneous voiding after treatment with metoclopramide for accompanying diabetic gastroparesis. Other reports of possible effects of metoclopramide therapy on the urinary tract have been limited. In an uncontrolled clinical trial in ten patients with a variety of chronic neurological conditions clinically characterized by flaccid, areflexic bladders, Vaidyanathan et al . . . [Full Text PDF of this Article]

Footnotes
Edited by Drummond Rennie, MD, Deputy Editor (West); Sharon Iverson, Assistant Editor.

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