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Jeffrey S. Dover, MD, FRCPC; Kenneth A. Arndt, MD

JAMA. 1992;268(3):342-344.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

This Contempo discussion will review human papillomavirus infection, dysplastic nevi as a precursor of melanoma, and extracorporeal photochemotherapy.

Warts, epidermal proliferations caused by the human papillomavirus (HPV), have an estimated incidence in young adults of 1.0%.¹ In the United States from 1966 to 1981, there was a sevenfold increase in the number of reported genital wart infections. At least 66 types of HPV have been reported to date; individual HPV types have different clinical manifestations and implications, but HPV type cannot be reproducibly predicted by morphologic criteria.² Human papillomavirus transmission usually occurs through skin contact, and warts can occur on any skin site, most commonly on the hands, feet, and genital skin. Genital warts, perhaps the most common sexually transmitted disease (if clinical, subclinical, and latent infections are considered), have at least a 50% chance of transmission of infection in a single sexual contact.

Human papillomavirus types 16,18, . . . [Full Text PDF of this Article]

Author Affiliations
New England Deaconess Hospital and Harvard Medical School, Boston, Mass; Beth Israel Hospital and Harvard Medical School, Boston, Mass

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