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David S. Siscovick, MD, MPH; Irena King, PhD; Sheila Weinmann, PhD; Rozenn Lemaitre, PhD, MPH; T. E. Raghunathan, PhD; Bruce Psaty, MD, PhD; Leonard Cobb, MD; Barbara Retzlaff, MPH, RD; Robert Knopp, MD
University of Washington Seattle

JAMA. 1996;275(11):836-837.

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—Dr Siguel suggests that consumption of seafood is not the primary dietary source of the long-chain n-3 PUFAs EPA and DHA. Among vegetarians and persons who consume low amounts of linoleic acid (C_18:2n-6), dietary intake of alphalinolenic acid (C_18:3n-3) from other foods such as tofu, soybean oil, and canola oil may increase cell membrane levels of EPA.¹

The purpose of our study was to assess the relation of the dietary intake and cell membrane levels of long-chain n-3 PUFAs derived from seafood and the risk of primary cardiac arrest in the community. In Seattle, Wash, seafood such as salmon, which is high in long-chain n-3 PUFAs, is an important component of the diet. As we noted, there was a strong correlation between dietary intake of long-chain n-3 PUFAs from seafood and red blood cell membrane long-chain n-3 fatty acid levels. The mean (SD) red blood . . . [Full Text PDF of this Article]

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