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Peggy Reynolds, PhD; Julie Von Behren, MPH
California Department of Health Services Emeryville

Elizabeth T. H. Fontham, PhD; Pelayo Correa, MD
Louisiana State University Medical Center New Orleans

Anna Wu, PhD
University of Southern California School of Medicine Los Angeles

Patricia A. Buffler, PhD
University of California School of Public Health Berkeley

Raymond S. Greenberg, MD, PhD
Medical University of South Carolina Charleston

JAMA. 1996;275(6):441-442.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—The article by Dr Hammond and colleagues,¹ reporting high measured levels of nicotine in occupational settings without smoking restrictions, even exceeding those from some household studies, underscores concerns about the health risks associated with workplace exposure to ETS. To the degree that nicotine serves as a marker of ETS, these data are consistent with the finding that point estimates for the relative risks associated with workplace sources of exposure were somewhat higher than those associated with adult exposures from a spouse or other household members in our study of ETS and lung cancer among women who were lifetime never-smokers.²

The evidence offered by Hammond et al regarding the potentially high historical levels of exposure to important constituents of ETS among nonsmoking workers in various settings without smoking restrictions prompted us to examine more closely the effect of workplace ETS exposure on risk of lung cancer . . . [Full Text PDF of this Article]

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