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John A. Ambrose, MD; Valentin Fuster, MD, PhD

JAMA. 1997;277(4):343-344.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Many therapeutic measures have been developed to effectively treat patients with coronary disease. While secondary prevention has become a vital component in maximizing reductions in coronary disease mortality, subsequent acute coronary events still occur and are often unexpected and unpredictable.

See also P 325.

We now recognize several standard risk factors for the development of coronary disease and a modulation of some of these risk factors has been shown to decrease the incidence and recurrence of coronary events.^1-3 With the help of noninvasive and invasive cardiologic technology, we may also stratify coronary patients into low- and high-risk subgroups.⁴ Mortality in coronary disease is directly related to the number of diseased vessels, the presence of left ventricular dysfunction, and an ischemic response on treadmill exercise testing, particularly when it occurs during the early stages of exercise. However, as stratification in prognosis is not all or none (ie, only some . . . [Full Text PDF of this Article]

Author Affiliations
From the Cardiovascular Institute, Mount Sinai Medical Center, New York, NY.

Footnotes
Reprints: John A. Ambrose, MD, Cardiovascular Institute, Mount Sinai Medical Center, New York, NY 10029-7462.

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