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JAMA. 1951;146(14):1275-1281. doi: 10.1001/jama.1951.03670140001001

AURICULAR FIBRILLATION

  1. Myron, M.D.;
  2. Robert Oblath, M.D.;
  3. Eliot Corday, M.D.;
  4. I. C. Brill, M.D.;
  5. H. E. Kruger;
  6. L. Allen Smith, M.D.;
  7. Joshua Fields, M.D.;
  8. Rexford Kennamer, M.D.;
  9. John A. Osborne, M.D.
  1. Los Angeles
  2. From the Institute for Medical Research, Cedars of Lebanon Hospital, and the Department of Medicine, University of California School of Medicine.

Since this article does not have an abstract, we have provided the first 150 words of the full text.

Excerpt

In past decades, many of the ablest medical investigators have attempted to resolve the mechanism of auricular fibrillation, the most important disturbance of the human heartbeat. The inconclusive nature of the results obtained by these workers undoubtedly is attributable to the fact that they did not utilize direct methods and their tools were not sufficiently sensitive to analyze the complex activity in the fibrillating auricles. For example, it it understandable that Sir James MacKenzie could not resolve the complex mechanism of auricular fibrillation from observations based solely on crude pulse tracings. The various earlier hypotheses were largely abandoned in 1920 when Sir Thomas Lewis, after extensive experimentation, advanced the circus movement theory. In the present investigation, the fibrillating auricle in man and animals has been reexamined with techniques not previously employed and apparatus not hitherto available. By these new methods the circus movement theory of auricular fibrillation has been found

Footnotes

  • Work by Dr. Oblath was performed during his tenure as research fellow of the American Heart Association.

  • This work was made possible by the financial support of the Beaumont Trust Fund (founded by Louis D. Beaumont).

  • Read before the Section on Internal Medicine at the Ninety-Ninth Annual Session of the American Medical Association, San Francisco, June 29 1950

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